What condition can zinc toxicity in aquatic animals lead to?

Zinc toxicity in aquatic animals primarily leads to neurologic signs due to the heavy metal's detrimental effects on the nervous system. Elevated zinc levels can disrupt the normal functioning of various physiological processes, including neurotransmitter function and cell signaling pathways. This often manifests as behavioral changes, coordination issues, and possible seizures, reflecting the neurotoxic impact of zinc.

In aquatic species, such as fish and invertebrates, exposure to elevated zinc concentrations can result from various sources, including contaminated water, sediment, or feed. As a result, monitoring zinc levels in aquatic ecosystems is essential to prevent adverse health effects in these animals.

While gastrointestinal distress is a concern in toxicities involving other substances, zinc's primary target is the nervous system. Similarly, excessive aggression is not a direct consequence of zinc toxicity; rather, behavioral changes may occur alongside a decline in overall health and neurological function. Lastly, improved respiratory function is not associated with zinc toxicity since heavy metals typically impair physiological functions rather than enhance them.